Impaired glucose tolerance in obese

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

In a multiethnic cohort of 162 obese youth with baseline impaired glucose tolerance followed prospectively for a mean of 2.9 years with standard-of-care dietary counseling, 65% reverted to normal gluc

Effect: improvement; 65% reversion rate (IGT to NGT)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)

Obese youth who reverted from IGT to NGT exhibited a 4-fold increase in oral disposition index, reflecting dynamic beta-cell hyper-responsiveness, while those who persisted with IGT or progressed to T

Effect: improvement; 4-fold increase in oDI (from 0.94 to 3.90)