progression from normal glucose tolerance to impaired glucose tolerance

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Ethnicity was not a statistically significant predictor of progression from normal glucose tolerance to impaired glucose tolerance in obese youth, unlike its strong role in predicting IGT reversion, w

Effect: null; OR not reported (p=NS)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)

Among 364 obese youth with normal glucose tolerance at baseline, 14% progressed to impaired glucose tolerance over 2.9 years, with progression characterized by a significant decline in beta-cell funct

Effect: decline; 14% progression rate (NGT to IGT)