Skin changes due to chronic exposure to non-ionizing radiation

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals chronically exposed to any of a range of LCSs — saccharin, sucralose, AceK, aspartame, or erythritol + aspartame — exhibited increased food consumption, lower post-prandial thermogenesis, incr

Effect: decline; saccharin-exposed rats had quadruple post-snack chow intake vs predictive group; 55% increase in visceral adipose mass with TFA+MSG+ASP vs TF

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in