ExploreOutcomeGlucose homeostasis
Outcome

Glucose homeostasis

Also known as: Glucose homeostasis (glucose tolerance, insulin sensitivity, fasting glucose, insulin levels, GLP-1 release)
3 findings 1 paper 3 related entities View in graph →

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Findings (27)

None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con
None
decline

Animals exposed to LCS exhibited impaired glucose tolerance, decreased insulin sensitivity, elevated fasting glucose, hyperinsulinemia, and impaired GLP-1 release — effects that sometimes occurred eve

Effect: decline; fasting glucose levels 1.6-fold higher in ASP-exposed vs controls; 2.25- and 2.3-fold higher for males and females in ASP+MSG vs controls; in

Size: fasting glucose levels 1.6-fold higher in ASP-exposed vs con

Papers (1)