HOMA-IR screening
Related entities
Findings (27)
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64
None
declineBaseline insulin resistance (HOMA-IR >2) was independently associated with failure to achieve sustained virological response during pegIFN-α-2a and ribavirin retreatment of HIV/HCV co-infected patient
Effect: decline; AOR 0.17; CI: 95% CI 0.05–0.64