HOMA-IR measurement

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models

Insulin resistance was a better predictor of sustained virological response than steatosis or cirrhosis in multivariable models, and is the only non-invasive measure among the three correlated predict

Effect: improvement; IR model had smaller -2 log likelihood value than steatosis or cirrhosis models