Stroke

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Intensive blood pressure control resulted in similar numbers of hemorrhagic and ischemic stroke subtypes compared to standard control in SPRINT, with no subtype showing a statistically significant tre

Effect: null; Hemorrhagic: 6 vs 7; LAA: 11 vs 13; CE: 11 vs 15; SAO: 8 vs 8

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78

Women with a history of hypertensive disorders of pregnancy had nearly 2.5 times the risk of subsequent chronic kidney disease compared to age- and parity-matched women with normotensive pregnancies.

Effect: decline; HR 2.41; CI: 95% CI 1.54-3.78